I've read that some diseases cause viral load to go down. Could it be that these bacteria are somehow more compelling to hiv than T-Cells and it attaches to them more promptly. Then, by virtue of the antibodies created by the human host to fight these other diseases, hiv is destroyed inadvertedly. When the disease is over, the remaining hiv is more likely to be more attracted to T-Cells because the remaining strains were the ones who were not attracted to the invading bacteria, and thus proceede to reproduce aggressively yielding the rebound in viral load observed. Maybe this natural decoy (if that turns out to be the explanation for the viral load decreases) added to aggressive viral load therapies during the infection would yield a very effective way to reduce viral load. What would have to be developed is harmless versions of these disease-causing bacteria that preserve their attractiveness to hiv.

Let's feed the monster till it bursts out!!!!

Your comments?

Interesting theory, but I don't think it works that way.